Pages that link to "Q24318945"

The following pages link to Intestinal apolipoprotein AI gene transcription is regulated by multiple distinct DNA elements and is synergistically activated by the orphan nuclear receptor, hepatocyte nuclear factor 4 (Q24318945):

Displayed 24 items.

• Hepatocyte nuclear factor 4 alpha (Q21114166) ‎ (← links)
• Hepatic nuclear factor 4, alpha (Q21496454) ‎ (← links)
• The nuclear receptors peroxisome proliferator-activated receptor alpha and Rev-erbalpha mediate the species-specific regulation of apolipoprotein A-I expression by fibrates (Q24315845) ‎ (← links)
• Hepatocyte nuclear factor 3/fork head homolog 11 is expressed in proliferating epithelial and mesenchymal cells of embryonic and adult tissues (Q24319881) ‎ (← links)
• Identification and characterization of a 315-base pair enhancer, located more than 55 kilobases 5' of the apolipoprotein B gene, that confers expression in the intestine (Q28594725) ‎ (← links)
• Genomic profiling of tumor necrosis factor alpha (TNF-alpha) receptor and interleukin-1 receptor knockout mice reveals a link between TNF-alpha signaling and increased severity of 1918 pandemic influenza virus infection (Q30394612) ‎ (← links)
• The SP1 sites of the human apoCIII enhancer are essential for the expression of the apoCIII gene and contribute to the hepatic and intestinal expression of the apoA-I gene in transgenic mice (Q33857168) ‎ (← links)
• Modification in Oxidative Stress, Inflammation, and Lipoprotein Assembly in Response to Hepatocyte Nuclear Factor 4α Knockdown in Intestinal Epithelial Cells (Q34412525) ‎ (← links)
• Hepatocyte nuclear factor 4alpha is essential for embryonic development of the mouse colon (Q36635519) ‎ (← links)
• Hepatocyte nuclear factor 4alpha in the intestinal epithelial cells protects against inflammatory bowel disease (Q36732009) ‎ (← links)
• Intestinal transcription and synthesis of apolipoprotein AI is regulated by five natural polymorphisms upstream of the apolipoprotein CIII gene (Q38346805) ‎ (← links)
• Characterization of a dominant negative mutant form of the HNF-4 orphan receptor (Q38354733) ‎ (← links)
• FOXO1 and LXRα downregulate the apolipoprotein A-I gene expression during hydrogen peroxide-induced oxidative stress in HepG2 cells (Q38728466) ‎ (← links)
• DNA binding and transcription activation specificity of hepatocyte nuclear factor 4 (Q39723950) ‎ (← links)
• TFIIB-directed transcriptional activation by the orphan nuclear receptor hepatocyte nuclear factor 4. (Q40018632) ‎ (← links)
• The A/G polymorphism in the -78 position of the apolipoprotein A-I promoter does not have a direct effect on transcriptional efficiency (Q41040042) ‎ (← links)
• Utilization of recombinant adenovirus and dominant negative mutants to characterize hepatocyte nuclear factor 4-regulated apolipoprotein AI and CIII expression (Q41109529) ‎ (← links)
• Human cholesteryl ester transfer protein gene proximal promoter contains dietary cholesterol positive responsive elements and mediates expression in small intestine and periphery while predominant liver and spleen expression is controlled by 5'-dist (Q41145439) ‎ (← links)
• Polyunsaturated fatty acyl coenzyme A suppress the glucose-6-phosphatase promoter activity by modulating the DNA binding of hepatocyte nuclear factor 4 alpha. (Q43895794) ‎ (← links)
• Functional domains of the nuclear receptor hepatocyte nuclear factor 4. (Q52196728) ‎ (← links)
• A hormone response element in the human apolipoprotein CIII (ApoCIII) enhancer is essential for intestinal expression of the ApoA-I and ApoCIII genes and contributes to the hepatic expression of the two linked genes in transgenic mice (Q73993872) ‎ (← links)
• Analysis of apolipoprotein A-I, lecithin:cholesterol acyltransferase and glucocerebrosidase genes in hypoalphalipoproteinemia (Q74238655) ‎ (← links)
• Estrogen regulation of the apolipoprotein AI gene promoter through transcription cofactor sharing (Q74424661) ‎ (← links)
• User:Daniel Mietchen/Scholia/To-do-lists/Works that are referenced and cited but do not have an identified author ‎ (← links | edit)